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366   Morphologic and Functional Evaluation of Immune Reconstitution in Tonsil Compared to Inguinal Lymphatic Tissue.

367   Longitudinal Monitoring of Chronically HIV-1-Infected Individuals for Cell-Associated Virus and Immune Responses.

368   Memory to Recall Antigen & CD4 T Cells Increasing After HAART Reside Within the Same CD4+ CD62L+ Compartment.

369   Quantitative Expression of Activation Markers on Defined CD4 and CD8 Lymphocyte Subsets Correlates with Viral Suppression/Relapse Status of HIV-1+ Patients Receiving HAART.

370   Reduction in TREC in HIV-1-Infected Individuals Is Not Explained by Increased Lymphocyte Proliferation Alone.

371   Modeling Anti-CD8 Depletion and Recovery of CD8+ T Cells in SIV-Infected Macaques.

372   T-Lymphocyte Changes after 3 Years of Controlled Viral Replication.

373   Pre-HIV-1 Seroconversion TREC Content May Be Predictive of Disease Progression.

374   Biphasic Changes in the Number of T-Cell Receptor Excisional Circles (TREC) in PBMC after Initiating Treatment of HIV-1 Infection with HAART.

375   No Substantial Changes either in TRECs or in CD45RA+ CD62L+ CD4+ Lymphocytes Following Treatment with Interleukin-2 in Advanced HIV.

376   IL-7 Prevents Apoptosis of Thymic Progenitor Cells Induced by HIV and Contributes to T Cell Reconstitution in the Thymus.

377   IL-7 Receptor (CD127) Expression on CD8 T-Cells Is Down Regulated by TNF-α but not IL-7, IL-10 or gp120.

378   Limited Thymic Contribution in CD4 T Cell Restoration during Early HAART.

379   Increased CD4+ T Cell Proliferation in HIV Infection Is Not Primarily Homeostatic and Reflects T Cell Activation Induced by Virus Replication.

380   CD4+ T Lymphocyte Nadir and the Effect of HAART on T Lymphocyte Restoration and Function in HIV Infection.

381   Blood Viral Load and Restoration of Gut CD4 T Cells in AIDS Patients with HAART: Influence of Opportunistic Intestinal Pathogens.

382   Relative Significance of Different Pathways of Immune Reconstitution in HIV-1 Infection as Estimated by Mathematical Modeling.