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Session 86 Poster Abstracts
Pharmacology of Protease Inhibitors
Tuesday, 1:30 - 3:30 pm
Poster Hall


613
Relationship between Lopinavir Concentration and Changes in Lipid Levels at 24 Weeks
B Best*1, S May1, M Witt2, C Kemper3, R Larsen4, C Diamond5, P Heseltine6, F He1, E Capparelli1, A McCutchan1, R Haubrich1, and the California Collaborative Treatment Group (CCTG)
1Univ. of California, San Diego, USA; 2Harbor-UCLA Med. Ctr., Univ. of California, Los Angeles Sch. of Med., Torrance, CA, USA; 3Santa Clara Valley Med. Ctr., San Jose, CA, USA; 4Univ. of Southern California, Los Angeles, USA; 5Univ. of California, Irvine, USA; and 6Quest Diagnostics, San Juan Capistrano, CA, USA

Background:  The relationship between LPV concentration and cholesterol elevation is not clear. The objective of this analysis was to explore associations between LPV concentration metrics and change in lipid values.

Methods:  Data were analyzed from CCTG 578, an ongoing, randomized, 3x2 factorial study of 3 adherence interventions crossed with therapeutic drug monitoring. LPV and RTV levels were drawn pre-, 2- and 4-hours post a witnessed week 2 LPV dose from naïve or experienced patients. Concentration data from the study was used to develop a population pharmacokinetic model. Post-hoc Bayesian estimates of individual subject’s LPV exposure measures were calculated (C2, C4, and C12 for estimated 2, 4, and 12 hour concentrations). Fasting lipids (total cholesterol (TC), HDL, LDL and triglycerides) were measured at day 0 and week 24.

Results:  For the 37 patients, the average log10 baseline HIV RNA and CD4 were 5.0 and 172. Mean (SD) 2, 4, and 12 hour LPV levels in mg/mL were 6.9 (3.5), 8.1 (3.5), and 5.6 (3.0). Baseline to week 24 TC increased from 157 to 202 while triglycerides increased from 196 to 317. Neither LPV nor RTV levels were associated with triglycerides, LDL or HDL changes. Increasing LPV concentrations (both C2 and C4) were associated with lower changes in week 24 TC (p<0.02). LPV C12 and RTV C2 and C4 showed trends in the same direction (p <0.15), but were less predictive. Multivariate models, controlling for gender, continued to demonstrate the same LPV effect; for every one mcg/ml increase in LPV C2, the average week 24 change in TC was 5.9 mg/dL less (p = 0.008).

Alternative models (2 piecewise linear forms) were explored due to a suggestion of differential effects of LPV concentration on TC depending on LPV concentration (initial increase in TC up to a level of 7 mg/mL and then decrease in TC as LPV concentration rose). In this model, increasing LPV levels (up to 7 mg/mL) were accompanied by non-significant TC increases (p = 0.7) while increasing LPV levels at higher concentrations (above 7 mg/mL) were significantly related to TC reductions (p = 0.005).

Conclusions:  In contrast to previous studies, this analysis did not find that higher LPV or RTV levels were accompanied by greater lipid increases. In fact, patients with higher LPV concentrations had smaller TC increases at week 24. LPV levels 2 and 4 hours after a witnessed dose were the best predictors while trough (C12) was not predictive of lipid changes.

Keywords: Lopinavir; lipid levels; concentration response relationship