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Session 86  Poster Abstracts
HCV and OIs in NeuroAIDS
Tuesday, 1-2:30 pm
Poster Hall
      464 Neuradapt: A Prospective Study concerning HIV-related Neurocognitve Impairment
Matteo Vassallo*1, A Harvey Langton1, C Pradier1, S Ferrando1, J Cottalorda1, C Caissotti1, J Durant1, G Malandain2, S Chanalet3, and P Dellamonica1
1L`Archet Hospītal - CHU Nice - France; 2INRIA - Sophia Antipolis - France; and 3Pasteur Hospital - CHU Nice - France
      465

Does Acute HCV Infection Affect the Central Nervous System in HIV-1-infected Individuals?
Alan Winston*, L Garvey, E Scotney, J Allsop, E Thomson, V Grover, J Main, J Cox, M Wyelinska, and S Taylor-Robinson
Imperial Coll London, UK
      466

Hepatitis C Seropositivity Is Not a Risk Factor for Sensory Neuropathy in Patients with HIV Infection
Catherine Cherry*1,2,3, J McArthur4, J Affandi5, E Yunihastuti6, S Vanar7, A Kamarulzaman7, D Imran6, D Hooker1, S Wesselingh1,2,3, and P Price8
1Macfarlane Burnet Inst, Melbourne, Australia; 2Monash Univ, Melbourne, Australia; 3Alfred Hosp, Melbourne, Australia; 4Johns Hopkins Univ, Baltimore, MD, US; 5Murdoch Univ, Perth, Australia; 6Pokdisus HIV Care Clinic, Jakarta, Indonesia; 7Univ of Malaya, Kuala Lumpur, Malaysia; and 8Univ of Western Australia, Perth
      467

Marked Increase of Regulatory T Cells and HIV Viral Load in the Cerebrospinal Fluid Is Associated with NeuroAIDS Opportunistic Infections
B Parra1, Andres Zea-Vera*1, E Martinez2, G Libreros1, M Barbosa1, and C Pardo-Villamizar3
1Universidad del Valle, Cali, Valle del Cauca, Colombia; 2Hospital Universitario del Valle, Cali, Valle del Cauca, Colombia; and 3Johns Hopkins Univ, Baltimore, MD, US
    468a Suppression of Polyomavirus JC Replication by Soluble Factors from Activated T Cells: Implications for Progressive Multifocal Leukoencephalopathy Therapy
L Sumibcay1, T Johnson2, A Nath2, and Vivek Nerurkar*1
1Univ of Hawaii, Honolulu, US and 2Johns Hopkins Univ, Baltimore, MD, US
    468b JC Virus VP1 from Cerebrospinal Fluid and Plasma of Patients with Progressive Multifocal Leukoencephalopathy Carry Specific Mutations of Aminoacid Residues Involved in Sialic Acid Binding
Paola Cinque*1, C Reid2, M Testa1, P Carmillo2, A Pazzi1, A Lugovskoy2, A Lazzarin1, and L Gorelik2
1San Raffaele Sci Inst, Milan, Italy and 2BiogenIDEC Inc, Cambridge, MA, US